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TESTICULAR CANCER INR   0 INR  0
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TESTICULAR CANCER

Testicular cancer occurs in the testicles (testes), which are located inside the scrotum, a loose bag of skin underneath the penis. The testicles produce male sex hormones and sperm for reproduction. Testicular cancer is highly treatable, even when cancer has spread beyond the testicle. Depending on the type and stage of testicular cancer, you may receive one of several treatments, or a combination. Symptoms • A n enlarged testicle or a small lump or area of hardness are the first signs of testicular cancer • A feeling of heaviness in the scrotum • A dull ache in the abdomen or groin • A sudden collection of fluid in the scrotum • Pain or discomfort in a testicle or the scrotum • Enlargement or tenderness of the breasts • Back pain Cancer usually affects only one testicle. Causes It's not clear what causes testicular cancer in most cases. Doctors know that testicular cancer occurs when healthy cells in a testicle become altered. Healthy cells grow and divide in an orderly way to keep your body functioning normally. But sometimes some cells develop abnormalities, causing this growth to get out of control — these cancer cells continue dividing even when new cells aren't needed. The accumulating cells form a mass in the testicle. Nearly all testicular cancers begin in the germ cells — the cells in the testicles that produce immature sperm. What causes germ cells to become abnormal and develop into cancer isn't known. Risk factors Factors that may increase your risk of testicular cancer include: • An undescended testicle (cryptorchidism). The testes form in the abdominal area during fetal development and usually descend into the scrotum before birth. Men who have a testicle that never descended are at greater risk of testicular cancer than are men whose testicles descended normally. • Abnormal testicle development. Conditions that cause testicles to develop abnormally, such as Klinefelter syndrome, may increase your risk of testicular cancer. • Family history. If family members have had testicular cancer, you may have an increased risk. • Age. Testicular cancer affects teens and younger men, particularly those between ages 15 and 35. However, it can occur at any age. • Race. Testicular cancer is more common in white men than in black men. Prevention There's no way to prevent testicular cancer. Self-examinations to identify testicular cancer at its earliest stage. Diagnosis In some cases men discover testicular cancer themselves, either unintentionally or while doing a testicular self-examination to check for lumps. In other cases, your doctor may detect a lump during a routine physical exam. To determine whether a lump is testicular cancer, your doctor may recommend: • Ultrasound. An ultrasound test can help your doctor determine the nature of any testicular lumps, such as whether the lumps are solid or fluid-filled. An ultrasound also tells your doctor whether lumps are inside or outside of the testicle. • Blood tests Type of cancer Your extracted testicle will be analyzed to determine the type of testicular cancer. The type of testicular cancer you have determines your treatment and your prognosis. In general, there are two types of testicular cancer: • Seminoma. Seminoma tumors occur in all age groups, but if an older man develops testicular cancer, it is more likely to be seminoma. Seminomas, in general, aren't as aggressive as nonseminomas. • Nonseminoma. Nonseminoma tumors tend to develop earlier in life and grow and spread rapidly. Several different types of nonseminoma tumors exist, including choriocarcinoma, embryonal carcinoma, teratoma and yolk sac tumor. Staging the cancer Once your doctor confirms your diagnosis, the next step is to determine the extent (stage) of the cancer. To determine whether cancer has spread outside of your testicle, you may undergo: • Computerized tomography (CT) scan • Blood tests. After these tests, your testicular cancer is assigned a stage. The stage helps determine what treatments are best for you. The stages of testicular cancer are indicated by Roman numerals that range from 0 to III, with the lowest stages indicating cancer that is limited to the area around the testicle. By stage III, the cancer is considered advanced and may have spread to other areas of the body, such as the lungs. Treatment The options for treating testicular cancer depend on several factors, including the type and stage of cancer, overall health. • Surgery to remove your testicle (radical inguinal orchiectomy) is the primary treatment for nearly all stages and types of testicular cancer. • Surgery to remove nearby lymph nodes (retroperitoneal lymph node dissection) • Radiation therapy: Radiation therapy uses high-powered energy beams, such as X-rays, to kill cancer cells. Radiation therapy is a treatment option that's sometimes used in people who have the seminoma type of testicular cancer. • Chemotherapy treatment uses drugs to kill cancer cells. Chemotherapy drugs travel throughout your body to kill cancer cells that may have migrated from the original tumor. Side effects of chemotherapy are depend on the specific drugs being used. Ask your doctor what to expect. Common side effects include • Fatigue, nausea, hair loss and an increased risk of infection. There are medications and treatments available that reduce some of the side effects of chemotherapy. • Chemotherapy may also lead to infertility in some men, which can be permanent in some cases. Talk to your doctor about your options for preserving your sperm before beginning chemotherapy. TESTICULAR CANCER TREATMENT IN JALANDHAR

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ENDOMETRIOSIS INR   0 INR  0
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ENDOMETRIOSIS

Endometriosis is a painful disorder in which tissue that normally lines the inside of your uterus — the endometrium — grows outside your uterus. Endometriosis most commonly involves your ovaries, fallopian tubes and the tissue lining your pelvis. Rarely, endometrial tissue may spread beyond pelvic organs. With endometriosis, displaced endometrial tissue continues to act as it normally would — it thickens, breaks down and bleeds with each menstrual cycle. Because this displaced tissue has no way to exit your body, it becomes trapped. When endometriosis involves the ovaries, cysts called endometriomas may form. Surrounding tissue can become irritated, eventually developing scar tissue and adhesions — abnormal bands of fibrous tissue that can cause pelvic tissues and organs to stick to each other. Endometriosis can cause pain — sometimes severe — especially during your period. Fertility problems also may develop. Fortunately, effective treatments are available. Symptoms The primary symptom of endometriosis is pelvic pain, often associated with your menstrual period. Although many women experience cramping during their menstrual period, women with endometriosis typically describe menstrual pain that's far worse than usual. They also tend to report that the pain increases over time. Common signs and symptoms of endometriosis may include: • Pelvic pain • Painful periods (dysmenorrhea) • Pain with intercourse • Pain with bowel movements or urination. • Excessive bleeding • Infertility. Endometriosis is first diagnosed in some women who are seeking treatment for infertility. • fatigue • diarrhea • constipation • bloating or nausea, especially during menstrual periods. The severity of your pain isn't necessarily a reliable indicator of the extent of the condition. Some women with mild endometriosis have intense pain, while others with advanced endometriosis may have little pain or even no pain at all. Endometriosis is sometimes mistaken for other conditions that can cause pelvic pain, such as pelvic inflammatory disease (PID) or ovarian cysts. It may be confused with irritable bowel syndrome (IBS), a condition that causes bouts of diarrhea, constipation and abdominal cramping. IBS can accompany endometriosis, which can complicate the diagnosis. Causes Although the exact cause of endometriosis is not certain, possible explanations include: • Retrograde menstruation. In retrograde menstruation, menstrual blood containing endometrial cells flows back through the fallopian tubes and into the pelvic cavity instead of out of the body. These displaced endometrial cells stick to the pelvic walls and surfaces of pelvic organs, where they grow and continue to thicken and bleed over the course of each menstrual cycle. • Transformation of peritoneal cells. In what's known as the "induction theory," experts propose that hormones or immune factors promote transformation of peritoneal cells — cells that line the inner side of your abdomen — into endometrial cells. • Embryonic cell transformation. Hormones such as estrogen may transform embryonic cells — cells in the earliest stages of development — into endometrial cell implants during puberty. • Surgical scar implantation. After a surgery, such as a hysterectomy or C-section, endometrial cells may attach to a surgical incision. • Endometrial cells transport. The blood vessels or tissue fluid (lymphatic) system may transport endometrial cells to other parts of the body. • Immune system disorder. It's possible that a problem with the immune system may make the body unable to recognize and destroy endometrial tissue that's growing outside the uterus. Risk factors Several factors place you at greater risk of developing endometriosis, such as: • Never giving birth • Starting your period at an early age • Going through menopause at an older age • Short menstrual cycles — for instance, less than 27 days • Having higher levels of estrogen in your body or a greater lifetime exposure to estrogen your body produces • Low body mass index • Alcohol consumption • One or more relatives (mother, aunt or sister) with endometriosis • Any medical condition that prevents the normal passage of menstrual flow out of the body • Uterine abnormalities Endometriosis usually develops several years after the onset of menstruation (menarche). Signs and symptoms of endometriosis end temporarily with pregnancy and end permanently with menopause, unless you're taking estrogen. Complications • Impaired fertility. Diagnosis • Pelvic exam • Transvaginal ultrasound • Laparoscopy. Treatment • Medications : Pain medications • Hormone therapy • Progestin therapy • Surgery

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Radical Hysterectomy INR   0 INR  0
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Radical Hysterectomy

Radical Hysterectomy surgery at Bhargava Hospital Advanced Gyne Surgery Cancer Center includes removal of the uterus, tubes and ovaries of both the sides (ovaries may be spared in young women), upper half of vagina , parametric (most of cardinal and uterosacral ligaments) and the draining primary cervical lymph nodes ( parametric ), obturator, internal and external iliac groups , and sometimes common iliac. Sacral group is not removed . Parametric lymph node is sampled and sent for frozen section biopsy , radiation therapy is to be considered if lymph nodes found involved. Radical Hysterectomy surgery in Jalandhar Radical Hysterectomy Doctor in Jalandhar

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Surgical Oncology INR   0 INR  0
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Surgical Oncology

Surgical Oncology is the oldest but still the most effective modality for treatment of solid cancers. In more than 50% patients surgery is often the only therapy necessary. For all regions from head to toe oncologically complete surgery remains the primary modality of management of cancer.

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Cytoreduction Surgery for Ovarian Cancer

Cytoreduction literally means a reduction in the number of cells. So cytoreductive surgery refers to a treatment of advanced ovarian cancer in which surgery attempts to remove as many cancerous cells as possible. Cytoreduction can also be: Primary - The first part of cancer treatment. Interval - Done after some chemotherapy has been given. Secondary - Done after someone has had a recurrence of their cancer. Tertiary - Done after several recurrences of the cancer

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